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A gross oversimplification of pathogenic processes that may index distinct neurobiological processes33 and contribute to situation specificity. Future research will need to identify distinct neurobiological substrates that contribute to individual differences in AIS.33,34 For example, previous clinical and developmental investigations have considered constructs similar to AIS (e.g., emotional lab
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Eatures are influenced by individual differences in neurobiology, personal experiences, and contextual characteristics.NIH-PA Author Manuscript NIH-PA Author Manuscript NIH-PA Author ManuscriptAcknowledgmentsThis research was supported in part by NIMH 1K01MH073717-01A2 (DAGD)NIH Public AccessAuthor ManuscriptMem Cognit. Author manuscript; available in PMC 2013 January 24.Published in final e
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Duction varied and increased with the density of agents per unitDuction varied and increased with the density of agents per unit cell; ii) the foraging strategy types that constituted an emerging polymorphism under clonal reproduction varied with runs, but showed some repeatable and some variable characteristics (e.g. at the very low densities of 2 to 10 agents, the movement-threshold parame
Sistency, routine, socialization) are the progenitors of AIS reactions in home versus school settings,8 which is well-illustrated in our results. For example, mothers describe their offspring with AIS as "difficult as a toddler," whereas teachers (who have spent a lot less time with the youth) do not. As parents are typically responsible for seeking intervention, it is not surprising that mother-
The rise of CSC theory, some specific CSC markers were being discovered to identify putative CSCs, and ALDH1 is becoming to act as a CSC marker in the CSC studies of variable tumor types in vitro and in vivo. Epithelial-to-mesenchymal transition (EMT) is an important driver of tumor invasion and metastasis, which may be a feature of CSC. Compared to ALDH1(-) EMT cells, only ALDH1(+) EMT cells had

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